Ultra-LEAF™ Purified anti-mouse CD200R (OX2R) Antibody, CD200R, OX-110,BioLegend,123917

Additional applications (for the relevant formats) include:in vivostudies3. The Ultra-LEAF™ purified antibody is recommended for functional assays.

Host

Rat

Reactivity

Mouse

Application

FC - Quality testedFA - Reported in the literature, not verified in house

Platform ID

BAB075042107

BioLegend

Headquarters

8999 BioLegend Way San Diego, CA 92121 United States

BioLegend

Contact

Tel: 1-858-455-9588
Fax: +49 (4131) 7023913

Email:

Product Specifications
Scientific Background

Specifications

NameUltra-LEAF™ Purified anti-mouse CD200R (OX2R) Antibody, CD200R, OX-110
Cat. No.123917
HostRat
RRIDAB_2890680 (BioLegend Cat. No. 123917)AB_2890680 (BioLegend Cat. No. 123918)
IsotypeRat IgG2a, κ
ReactivityMouse
ApplicationFC - Quality testedFA - Reported in the literature, not verified in house
ClonalityMonoclonal
Clone NumberOX-110
ConcentrationThe antibody is bottled at the concentration indicated on the vial, typically between 2 mg/mL and 3 mg/mL. Older lots may have also been bottled at 1 mg/mL. To obtain lot-specific concentration and expiration, please enter the lot number in ourCertificate of Analysisonline tool.
TargetCD200R
ImmunogenFusion protein mouse CD200RCD4d3
PurityThe Ultra-LEAF™ (Low Endotoxin, Azide-Free) antibody was purified by affinity chromatography.
Formulation0.2 µm filtered in phosphate-buffered solution, pH 7.2, containing no preservative.
StorageThe antibody solution should be stored undiluted between 2°C and 8°C. This Ultra-LEAF™ solution contains no preservative; handle under aseptic conditions.
Regulatory StatusResearch Use Only

Scientific Background

CD200R, also known as OX2R, is a membrane glycoprotein with up to 70% of its weight derived from N-linked glycosylation. CD200R is expressed primarily by monocytes and neutrophils but also by other leukocytes including T lymphocytes and mast cells. The interaction between CD200 and CD200R may contribute to pathways that suppress and limit macrophage induced inflammatory damage in tissue. Studies suggest that CD200-CD200R interaction may be involved in the control of myeloid cellular function. Levels of expression on resting peripheral blood cells are relatively low.

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