Search results for P16-INK4A

p16-INK4A (pS7) Antibody, Abbexa, abx031885

This product is currently in development. The lead time for this product may be several months. Please contact us at [email protected] for an updated lead time before purchasing this product. This product is currently in development. The lead time for this product may be several months. Please contact us at [email protected] for an updated lead time before purchasing this product. p16-INK4A functions as a stabilizer of the tumor suppressor protein p53 as it can interact with, and sequester, MDM1, a protein responsible for the degradation of p53. This protein acts as a negative regulator of the proliferation of normal cells by interacting strongly with CDK4 and CDK6. This inhibits their ability to interact with cyclins D and to phosphorylate the retinoblastoma protein. The gene for this protein is frequently mutated or deleted in a wide variety of tumors, and is known to be an important tumor suppressor gene.

p16 INK4A (D7C1M) Rabbit Monoclonal Antibody (SignalFlex^TMmFluor^TMUV375 Conjugate)#92018, Cell Signaling Technology (CST), 92018

p16 INK4A (D7C1M) Rabbit mAb (SignalFlex^TM mFluor^TM UV375 Conjugate) recognizes endogenous levels of total p16 INK4A protein. This antibody does not cross-react with p15 INK4B. This antibody is not recommended for use in immunohistochemistry.

p16 INK4A (D7C1M) Rabbit Monoclonal Antibody (SignalFlex^TMAlexa Fluor^®350 Conjugate)#23818, Cell Signaling Technology (CST), 23818

p16 INK4A (D7C1M) Rabbit mAb (SignalFlex^TM Alexa Fluor^®350 Conjugate) recognizes endogenous levels of total p16 INK4A protein. This antibody does not cross-react with p15 INK4B. This antibody is not recommended for use in immunohistochemistry.

p16 INK4A (D7C1M) Rabbit Monoclonal Antibody (SignalFlex^TMAlexa Fluor^®555 Conjugate)#84411, Cell Signaling Technology (CST), 84411

p16 INK4A (D7C1M) Rabbit mAb (SignalFlex^TM Alexa Fluor^®555 Conjugate) recognizes endogenous levels of total p16 INK4A protein. This antibody does not cross-react with p15 INK4B. This antibody is not recommended for use in immunohistochemistry.

p16 INK4A (D7C1M) Rabbit Monoclonal Antibody (SignalFlex^TMAlexa Fluor^®700 Conjugate)#74405, Cell Signaling Technology (CST), 74405

p16 INK4A (D7C1M) Rabbit mAb (SignalFlex^TM Alexa Fluor^®700 Conjugate) recognizes endogenous levels of total p16 INK4A protein. This antibody does not cross-react with p15 INK4B. This antibody is not recommended for use in immunohistochemistry.

p16 INK4A (D7C1M) Rabbit Monoclonal Antibody (SignalFlex^TMmFluor^TMRed 780 Conjugate)#25439, Cell Signaling Technology (CST), 25439

p16 INK4A (D7C1M) Rabbit mAb (SignalFlex^TM mFluor^TM Red 780 Conjugate) recognizes endogenous levels of total p16 INK4A protein. This antibody does not cross-react with p15 INK4B. This antibody is not recommended for use in immunohistochemistry.

p16 INK4A (D7C1M) Rabbit Monoclonal Antibody (SignalFlex^TMmFluor^TMViolet 510 Conjugate)#23380, Cell Signaling Technology (CST), 23380

p16 INK4A (D7C1M) Rabbit mAb (SignalFlex^TM mFluor^TM Violet 510 Conjugate) recognizes endogenous levels of total p16 INK4A protein. This antibody does not cross-react with p15 INK4B. This antibody is not recommended for use in immunohistochemistry.

p16 INK4A Rabbit pAb- ABclonal, ABclonal, A23882

p16-INK4a (Phospho-Ser152) Antibody, Abbexa, abx012705

Rabbit polyclonal antibody against p16-INK4a protein. Immunogen region is C-terminal. Specificity is as follows for the reactive species: H:S152.

Cyclin-Dependent Kinase Inhibitor 2A / P16-INK4A Antibody, Abbexa, abx236064

CDKN2A / P16-INK4A Antibody is a Rabbit Polyclonal against CDKN2A. CDKN2A generates several transcript variants which differ in their first exons. At least three alternatively-spliced variants encoding distinct proteins have been reported, and two are related, named p16-INK4 and p14 respetively, sharing 50% identity. The third one is completely stuctually un-related, called p14(ARF) or p19(ARF). This antibody reacts with p16 specifically. P16 plays an important role in regulating the cell cycle, and mutations in p16 increase the risk of developing a variety of cancers, notably melanoma.